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BiologicalX

Comparison

Armodafinil vs CJC-1295

Side-by-side of Armodafinil and CJC-1295. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Armodafinil

  • FDA approved in 2007 for narcolepsy, shift-work sleep disorder, and OSA residual sleepiness
  • R-enantiomer of modafinil; 150 mg armodafinil is roughly equivalent to 200 mg modafinil
  • Schedule IV controlled in the US; prescription-only globally
  • Longer terminal half-life of about 15 hours produces extended late-day wakefulness coverage
  • Same CYP3A4 induction as modafinil; reduces hormonal contraceptive efficacy
  • Side-effect profile and dermatologic risk warnings mirror modafinil

CJC-1295

  • GHRH analog that binds the GHRH receptor on pituitary somatotrophs to release endogenous GH
  • DAC variant has ~7 day half-life via albumin binding; non-DAC variant ~30 minutes
  • Teichman 2006 trial showed sustained 2 to 10 fold IGF-1 elevation at 60 to 250 mcg/kg DAC dosing
  • Anecdotal protocols pair non-DAC CJC-1295 with Ipamorelin to mimic pulsatile GH release
  • Side effects: water retention, numbness or tingling at injection site, vivid dreams, transient flushing
  • No completed phase III RCTs; research-use-only and not FDA approved

Side-by-side

Attribute Armodafinil CJC-1295
Category pharmaceutical peptide
Also known as Nuvigil, R-modafinil, (R)-(-)-modafinil CJC-1295 DAC, CJC-1295 no-DAC, Mod GRF 1-29, tesamorelin analog
Half-life (hr) 15 168
Typical dose (mg) 150 0.1
Dosing frequency daily, morning weekly (DAC); 1-3x daily (non-DAC)
Routes oral subcutaneous
Onset (hr) 1 1
Peak (hr) 3 3
Molecular weight 273.35 3367.83
Molecular formula C15H15NO2S C152H252N44O42
Mechanism Weak dopamine reuptake inhibition plus downstream activation of histaminergic, noradrenergic, and orexinergic wake systems; R-enantiomer of modafinil with longer half-life. Binds the GHRH receptor on pituitary somatotrophs, stimulating pulsatile growth-hormone release. The DAC modification extends plasma residence by tethering the peptide to serum albumin via a maleimide-cysteine bond.
Legal status Schedule IV (US); prescription-only globally; not a supplement Not FDA approved; research-use-only grey market; banned by WADA
WADA status banned banned
DEA / Rx Schedule IV Not FDA approved; not scheduled; research-chemical status
Pregnancy Not recommended Insufficient data; not recommended
CAS 112111-43-0 446262-90-4
PubChem CID 9148206 91971820
Wikidata Q4791953 Q5012154

Safety profile

Armodafinil

Common side effects

  • headache
  • nausea
  • dizziness
  • anxiety
  • insomnia (with later-day dosing)
  • dry mouth
  • mild blood pressure elevation

Contraindications

  • recent myocardial infarction
  • unstable angina
  • left ventricular hypertrophy
  • significant arrhythmia
  • history of Stevens-Johnson syndrome
  • psychotic disorders
  • pregnancy
  • concurrent MAOI use

Interactions

  • hormonal contraceptives: CYP3A4 induction reduces contraceptive efficacy; use barrier method(major)
  • cyclosporine: reduced cyclosporine levels via CYP3A4 induction(major)
  • warfarin: CYP2C9 inhibition raises INR(moderate)
  • phenytoin: CYP2C19 inhibition raises phenytoin levels(moderate)
  • MAOIs: potential hypertensive reaction(major)
  • classical stimulants: additive cardiovascular and sleep-disruption effects(moderate)

CJC-1295

Common side effects

  • injection-site reactions
  • water retention
  • numbness or tingling at injection site
  • vivid dreams
  • transient flushing
  • head pressure or mild headache

Contraindications

  • pregnancy
  • active malignancy
  • diabetic retinopathy (theoretical)
  • history of pituitary tumor

Interactions

  • Ipamorelin: synergistic GH release; commonly co-administered in anecdotal protocols(minor)
  • insulin: GH-induced insulin resistance can shift glycemic control over weeks(moderate)
  • corticosteroids: blunt GH-axis response; reduce expected efficacy(moderate)

Which Should You Take?

Armodafinil comes out ahead for most readers on the criteria we weight: 3 catalogued goals, controlled substance, oral dosing, with a Tier-A outcome catalogued. CJC-1295 is the right call when one of the conditionals below applies.

  • If your priority is wakefulness, pick Armodafinil.
  • If your priority is focus or working memory, pick Armodafinil.
  • If your priority is post-training recovery, pick CJC-1295.
  • If your priority is growth-hormone axis, pick CJC-1295.

Edge case: If you cannot self-administer injections, Armodafinil is the only oral option in this pair.

Default choice: Armodafinil. Wider use case, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for CJC-1295 only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Armodafinil and CJC-1295?

Armodafinil and CJC-1295 differ in category (pharmaceutical vs peptide), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Armodafinil or CJC-1295?

Armodafinil half-life is 15 hours; CJC-1295 half-life is 168 hours.

Can you stack Armodafinil with CJC-1295?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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