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Comparison

CJC-1295 vs Clomiphene

Side-by-side of CJC-1295 and Clomiphene. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

CJC-1295

  • GHRH analog that binds the GHRH receptor on pituitary somatotrophs to release endogenous GH
  • DAC variant has ~7 day half-life via albumin binding; non-DAC variant ~30 minutes
  • Teichman 2006 trial showed sustained 2 to 10 fold IGF-1 elevation at 60 to 250 mcg/kg DAC dosing
  • Anecdotal protocols pair non-DAC CJC-1295 with Ipamorelin to mimic pulsatile GH release
  • Side effects: water retention, numbness or tingling at injection site, vivid dreams, transient flushing
  • No completed phase III RCTs; research-use-only and not FDA approved

Clomiphene

  • SERM that blocks estrogen-receptor negative feedback at the hypothalamus, raising LH and FSH
  • FDA approved 1967 for ovulation induction in anovulatory women at 50 to 100 mg cycle days 5 to 9
  • Off-label in men at 12.5 to 25 mg daily raises endogenous testosterone while preserving fertility
  • Enclomiphene (trans-isomer) is preferred for male use; cleaner PK and less estrogenic side effect burden
  • Visual disturbances occur in ~1 to 2% of users; persistent symptoms warrant immediate cessation
  • Letrozole has displaced clomiphene as first-line ovulation induction in PCOS (Legro 2014)

Side-by-side

Attribute CJC-1295 Clomiphene
Category peptide pharmaceutical
Also known as CJC-1295 DAC, CJC-1295 no-DAC, Mod GRF 1-29, tesamorelin analog Clomid, clomiphene citrate, Serophene, enclomiphene
Half-life (hr) 168 168
Typical dose (mg) 0.1 25
Dosing frequency weekly (DAC); 1-3x daily (non-DAC) 5-day pulse cycle days 5 to 9 (women); daily or every other day (men, off-label)
Routes subcutaneous oral
Onset (hr) 1 6
Peak (hr) 3 7
Molecular weight 3367.83 405.96
Molecular formula C152H252N44O42 C26H28ClNO
Mechanism Binds the GHRH receptor on pituitary somatotrophs, stimulating pulsatile growth-hormone release. The DAC modification extends plasma residence by tethering the peptide to serum albumin via a maleimide-cysteine bond. Selective estrogen receptor modulator that antagonizes estrogen at the hypothalamus and pituitary, increasing GnRH and gonadotropin output, which drives gonadal steroidogenesis.
Legal status Not FDA approved; research-use-only grey market; banned by WADA Prescription only (FDA approved for ovulation induction; off-label in men)
WADA status banned banned
DEA / Rx Not FDA approved; not scheduled; research-chemical status Rx only (not a controlled substance)
Pregnancy Insufficient data; not recommended Category X; contraindicated in pregnancy
CAS 446262-90-4 911-45-5
PubChem CID 91971820 1548953
Wikidata Q5012154 Q416785

Safety profile

CJC-1295

Common side effects

  • injection-site reactions
  • water retention
  • numbness or tingling at injection site
  • vivid dreams
  • transient flushing
  • head pressure or mild headache

Contraindications

  • pregnancy
  • active malignancy
  • diabetic retinopathy (theoretical)
  • history of pituitary tumor

Interactions

  • Ipamorelin: synergistic GH release; commonly co-administered in anecdotal protocols(minor)
  • insulin: GH-induced insulin resistance can shift glycemic control over weeks(moderate)
  • corticosteroids: blunt GH-axis response; reduce expected efficacy(moderate)

Clomiphene

Common side effects

  • hot flushes
  • mood changes
  • abdominal discomfort
  • breast tenderness
  • visual disturbances (rare)
  • headache

Contraindications

  • pregnancy
  • active liver disease
  • ovarian cysts (not PCOS-related)
  • uncontrolled thyroid or adrenal disorder
  • abnormal uterine bleeding of undetermined origin
  • hormone-sensitive cancer

Interactions

  • tamoxifen: competing SERM activity; not used together(moderate)
  • ospemifene: competing SERM activity(moderate)
  • anastrozole: additive estrogen reduction; sometimes combined in male protocols(minor)
  • TRT (exogenous testosterone): TRT suppresses HPT axis that clomiphene targets; do not combine(moderate)

Which Should You Take?

Clomiphene comes out ahead for most readers on the criteria we weight: 2 catalogued goals, prescription-only, oral dosing, with a Tier-A outcome catalogued. CJC-1295 is the right call when one of the conditionals below applies.

  • If your priority is post-training recovery, pick CJC-1295.
  • If your priority is growth-hormone axis, pick CJC-1295.
  • If your priority is hormonal optimization, pick Clomiphene.
  • If your priority is fertility, pick Clomiphene.

Edge case: If you cannot self-administer injections, Clomiphene is the only oral option in this pair.

Default choice: Clomiphene. Wider use case, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for CJC-1295 only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between CJC-1295 and Clomiphene?

CJC-1295 and Clomiphene differ in category (peptide vs pharmaceutical), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, CJC-1295 or Clomiphene?

CJC-1295 half-life is 168 hours; Clomiphene half-life is 168 hours.

Can you stack CJC-1295 with Clomiphene?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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