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Comparison

CJC-1295 vs Omega-3 (EPA/DHA)

Side-by-side of CJC-1295 and Omega-3 (EPA/DHA). Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

CJC-1295

  • GHRH analog that binds the GHRH receptor on pituitary somatotrophs to release endogenous GH
  • DAC variant has ~7 day half-life via albumin binding; non-DAC variant ~30 minutes
  • Teichman 2006 trial showed sustained 2 to 10 fold IGF-1 elevation at 60 to 250 mcg/kg DAC dosing
  • Anecdotal protocols pair non-DAC CJC-1295 with Ipamorelin to mimic pulsatile GH release
  • Side effects: water retention, numbness or tingling at injection site, vivid dreams, transient flushing
  • No completed phase III RCTs; research-use-only and not FDA approved

Omega-3 (EPA/DHA)

  • Reduces fasting triglycerides 20-50% at 2-4 g/day in hypertriglyceridemic patients
  • REDUCE-IT showed 25% relative risk reduction in major CV events at 4 g/day icosapent ethyl
  • Modest antidepressant effect (SMD ~0.40) for EPA-dominant formulations at 1-2 g/day
  • Atrial fibrillation incidence rises ~30-50% at 4 g/day; relevant for older patients with pre-existing CV disease
  • Tissue omega-3 index (RBC EPA + DHA) target ~8%; Western baseline typically 4-5%
  • Triglyceride and re-esterified triglyceride forms absorb ~70% better than ethyl esters in fasted state

Side-by-side

Attribute CJC-1295 Omega-3 (EPA/DHA)
Category peptide supplement
Also known as CJC-1295 DAC, CJC-1295 no-DAC, Mod GRF 1-29, tesamorelin analog fish oil, EPA, DHA, marine omega-3
Half-life (hr) 168 48
Typical dose (mg) 0.1 2000
Dosing frequency weekly (DAC); 1-3x daily (non-DAC) 1 to 2 times daily with food
Routes subcutaneous oral
Onset (hr) 1 4
Peak (hr) 3 12
Molecular weight 3367.83 302.45
Molecular formula C152H252N44O42 C20H30O2 (EPA); C22H32O2 (DHA)
Mechanism Binds the GHRH receptor on pituitary somatotrophs, stimulating pulsatile growth-hormone release. The DAC modification extends plasma residence by tethering the peptide to serum albumin via a maleimide-cysteine bond. Substitutes arachidonic acid in membrane phospholipids, shifting eicosanoid production toward less-inflammatory 3-series prostaglandins and 5-series leukotrienes. Activates PPAR-alpha to lower hepatic VLDL/triglyceride synthesis. DHA modulates synaptic membrane fluidity and neuronal function.
Legal status Not FDA approved; research-use-only grey market; banned by WADA Dietary supplement; prescription forms (icosapent ethyl, omega-3 acid ethyl esters) for severe hypertriglyceridemia
WADA status banned allowed
DEA / Rx Not FDA approved; not scheduled; research-chemical status Not scheduled
Pregnancy Insufficient data; not recommended Recommended at 200 to 600 mg DHA/day for fetal development
CAS 446262-90-4 10417-94-4
PubChem CID 91971820 446284
Wikidata Q5012154 Q207688

Safety profile

CJC-1295

Common side effects

  • injection-site reactions
  • water retention
  • numbness or tingling at injection site
  • vivid dreams
  • transient flushing
  • head pressure or mild headache

Contraindications

  • pregnancy
  • active malignancy
  • diabetic retinopathy (theoretical)
  • history of pituitary tumor

Interactions

  • Ipamorelin: synergistic GH release; commonly co-administered in anecdotal protocols(minor)
  • insulin: GH-induced insulin resistance can shift glycemic control over weeks(moderate)
  • corticosteroids: blunt GH-axis response; reduce expected efficacy(moderate)

Omega-3 (EPA/DHA)

Common side effects

  • fishy aftertaste
  • eructation (fish burps)
  • mild dyspepsia
  • loose stools at high doses

Contraindications

  • fish allergy (use algal omega-3 alternative)
  • active bleeding disorders
  • scheduled surgery (discontinue 5-7 days prior)

Interactions

  • warfarin and DOACs: additive antiplatelet effect at 2+ g/day; meaningful bleeding risk(moderate)
  • aspirin and antiplatelet agents: additive bleeding risk at high doses(moderate)
  • statins: complementary cardiovascular effects; no pharmacokinetic interaction(minor)
  • antiarrhythmics: high-dose omega-3 increases AF risk; relevant in pre-existing arrhythmia(moderate)

Which Should You Take?

Omega-3 (EPA/DHA) comes out ahead for most readers on the criteria we weight: 3 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-A outcome catalogued. CJC-1295 is the right call when one of the conditionals below applies.

  • If your priority is post-training recovery, pick CJC-1295.
  • If your priority is growth-hormone axis, pick CJC-1295.
  • If your priority is cardiovascular health, pick Omega-3 (EPA/DHA).
  • If your priority is healthspan extension, pick Omega-3 (EPA/DHA).

Edge case: If you want to avoid research-only / gray-market sourcing, Omega-3 (EPA/DHA) is the more accessible choice.

Default choice: Omega-3 (EPA/DHA). Lower friction to source, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for CJC-1295 only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between CJC-1295 and Omega-3 (EPA/DHA)?

CJC-1295 and Omega-3 (EPA/DHA) differ in category (peptide vs supplement), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, CJC-1295 or Omega-3 (EPA/DHA)?

CJC-1295 half-life is 168 hours; Omega-3 (EPA/DHA) half-life is 48 hours.

Can you stack CJC-1295 with Omega-3 (EPA/DHA)?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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