Skip to content
BiologicalX

Comparison

CJC-1295 vs PT-141

Side-by-side of CJC-1295 and PT-141. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

CJC-1295

  • GHRH analog that binds the GHRH receptor on pituitary somatotrophs to release endogenous GH
  • DAC variant has ~7 day half-life via albumin binding; non-DAC variant ~30 minutes
  • Teichman 2006 trial showed sustained 2 to 10 fold IGF-1 elevation at 60 to 250 mcg/kg DAC dosing
  • Anecdotal protocols pair non-DAC CJC-1295 with Ipamorelin to mimic pulsatile GH release
  • Side effects: water retention, numbness or tingling at injection site, vivid dreams, transient flushing
  • No completed phase III RCTs; research-use-only and not FDA approved

PT-141

  • Cyclic 7-amino-acid synthetic peptide and melanocortin receptor agonist (MC4R-preferring)
  • FDA approved in 2019 as Vyleesi for hypoactive sexual desire disorder in pre-menopausal women
  • Acts centrally on hypothalamic sexual-desire circuits rather than peripherally on vasculature
  • On-demand dosing: subcutaneous 1.75 mg approximately 45 minutes before sexual activity
  • Common adverse effects: nausea (~40%), flushing, headache, injection-site reactions, hyperpigmentation
  • Off-label male ED use is documented but not FDA approved; mechanism is distinct from PDE5 inhibitors

Side-by-side

Attribute CJC-1295 PT-141
Category peptide peptide
Also known as CJC-1295 DAC, CJC-1295 no-DAC, Mod GRF 1-29, tesamorelin analog Bremelanotide, Vyleesi
Half-life (hr) 168 2.7
Typical dose (mg) 0.1 1.75
Dosing frequency weekly (DAC); 1-3x daily (non-DAC) as needed (max once per 24 hours, max 8 per month)
Routes subcutaneous subcutaneous
Onset (hr) 1 0.75
Peak (hr) 3 1.5
Molecular weight 3367.83 1025.18
Molecular formula C152H252N44O42 C50H68N14O10
Mechanism Binds the GHRH receptor on pituitary somatotrophs, stimulating pulsatile growth-hormone release. The DAC modification extends plasma residence by tethering the peptide to serum albumin via a maleimide-cysteine bond. Synthetic agonist of melanocortin receptors with preference for MC4R, expressed in hypothalamic and limbic circuits regulating sexual motivation. Engages central pathways distinct from peripheral PDE5-mediated vasodilation.
Legal status Not FDA approved; research-use-only grey market; banned by WADA Prescription only as Vyleesi; FDA-approved 2019 for HSDD in pre-menopausal women. Compounded versions sold off-label for male sexual function are research-use-only grey market.
WADA status banned allowed
DEA / Rx Not FDA approved; not scheduled; research-chemical status Rx only (not a controlled substance) for the FDA-approved Vyleesi formulation
Pregnancy Insufficient data; not recommended Not recommended; contraindicated during pregnancy per Vyleesi label
CAS 446262-90-4 189691-06-3
PubChem CID 91971820 9941379
Wikidata Q5012154 Q422059

Safety profile

CJC-1295

Common side effects

  • injection-site reactions
  • water retention
  • numbness or tingling at injection site
  • vivid dreams
  • transient flushing
  • head pressure or mild headache

Contraindications

  • pregnancy
  • active malignancy
  • diabetic retinopathy (theoretical)
  • history of pituitary tumor

Interactions

  • Ipamorelin: synergistic GH release; commonly co-administered in anecdotal protocols(minor)
  • insulin: GH-induced insulin resistance can shift glycemic control over weeks(moderate)
  • corticosteroids: blunt GH-axis response; reduce expected efficacy(moderate)

PT-141

Common side effects

  • nausea (~40%)
  • flushing
  • headache
  • injection-site reactions
  • hyperpigmentation (focal, gums, face, breasts)
  • transient blood pressure increase (~6 mmHg systolic)

Contraindications

  • uncontrolled hypertension
  • established cardiovascular disease
  • pregnancy
  • naltrexone co-administration (reduces opioid efficacy due to MC receptor crosstalk)

Interactions

  • naltrexone (oral): bremelanotide reduces oral naltrexone exposure significantly; avoid co-administration(major)
  • antihypertensives: transient BP rise after bremelanotide can offset BP control(moderate)
  • PDE5 inhibitors (sildenafil, tadalafil): no documented adverse interaction; mechanisms are non-overlapping(minor)

Which Should You Take?

CJC-1295 and PT-141 score evenly on the criteria we weight (goal breadth, legal accessibility, evidence depth). The conditionals below should drive the decision more than any aggregate score.

  • If your priority is post-training recovery, pick CJC-1295.
  • If your priority is growth-hormone axis, pick CJC-1295.
  • If your priority is sexual function, pick PT-141.
  • If your priority is libido, pick PT-141.

Edge case: PT-141 is contraindicated in pregnancy; CJC-1295 is the safer pick if that applies.

Default choice: either is defensible. CJC-1295 edges out on goal breadth + legal accessibility; PT-141 is the right call if your priority sits in the goals listed above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between CJC-1295 and PT-141?

CJC-1295 and PT-141 differ in category (peptide vs peptide), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, CJC-1295 or PT-141?

CJC-1295 half-life is 168 hours; PT-141 half-life is 2.7 hours.

Can you stack CJC-1295 with PT-141?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

Go deeper