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BiologicalX

Comparison

DHEA vs Selank

Side-by-side of DHEA and Selank. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

DHEA

  • Adrenal androgen precursor; serum DHEA-S declines progressively after the third decade of life
  • OTC dietary supplement in US under DSHEA 1994; prescription in EU, UK, Canada, Australia
  • FDA approved as Intrarosa (6.5 mg vaginal insert) for postmenopausal dyspareunia in 2016
  • Acts as tissue-specific prohormone converted intracrinologically to testosterone and estrogens
  • Best evidence: adrenal insufficiency replacement and vaginal atrophy; weaker on cognition and longevity
  • WADA banned in competitive sport; banned in NCAA, MLB, NFL, IOC settings

Selank

  • Synthetic heptapeptide analog of tuftsin developed in Russia in the 1990s
  • Approved in Russia for generalized anxiety disorder and asthenic conditions
  • Russian RCTs report anxiolytic effects comparable to medazepam without sedation or dependence
  • Modulates GABAergic and serotonergic signaling and BDNF expression in preclinical models
  • Most commonly administered intranasally; subcutaneous use is anecdotal
  • No Western-validated trials; not FDA approved; research-use-only outside Russia

Side-by-side

Attribute DHEA Selank
Category hormone peptide
Also known as dehydroepiandrosterone, prasterone, Intrarosa TP-7, Tuftsin analog
Half-life (hr) 12 0.5
Typical dose (mg) 25 0.4
Dosing frequency daily, typically morning 2-3x daily (intranasal)
Routes oral, vaginal, topical intranasal, subcutaneous
Onset (hr) 1 0.25
Peak (hr) 1 1
Molecular weight 288.42 751.85
Molecular formula C19H28O2 C33H57N11O9
Mechanism Steroid prohormone converted intracrinologically to testosterone and estrogens in target tissues; also exerts direct effects via sigma-1 receptor, GABA-A modulation, and glucocorticoid receptor interaction. Modulates GABAergic, serotonergic, and dopaminergic signaling. Increases BDNF expression in hippocampal neurons in preclinical models. Modulates enkephalin levels and immune cytokine signaling via tuftsin-like activity.
Legal status OTC supplement in US (DSHEA 1994); prescription in EU, UK, Canada, Australia Approved as a prescription anxiolytic in Russia; not FDA approved; research-use-only grey market in most other jurisdictions
WADA status banned unknown
DEA / Rx OTC supplement in US (not scheduled); Rx in EU, UK, Canada, Australia Not FDA approved; not scheduled; research-chemical status outside Russia
Pregnancy Contraindicated in pregnancy Not recommended; insufficient data
CAS 53-43-0 129954-34-3
PubChem CID 5881 11765600
Wikidata Q411733 Q4416793

Safety profile

DHEA

Common side effects

  • acne
  • oily skin
  • hirsutism (women)
  • gynecomastia (men, higher doses)
  • irritability
  • insomnia

Contraindications

  • hormone-sensitive cancer (breast, ovarian, prostate)
  • active liver disease
  • uncontrolled lipid disorder
  • pregnancy and lactation

Interactions

  • warfarin: case reports of altered INR; monitor(moderate)
  • estrogens (HRT): additive estrogenic effect via conversion; monitor(moderate)
  • insulin: may improve insulin sensitivity slightly; monitor glucose(minor)
  • anastrozole: may reduce DHEA-derived estrogen; clinical relevance unclear(minor)

Selank

Common side effects

  • mild nasal irritation (intranasal)
  • transient drowsiness (uncommon)
  • mild headache

Contraindications

  • pregnancy
  • lactation
  • severe psychiatric disorder (insufficient data)

Interactions

  • benzodiazepines: additive anxiolytic effect; potential for over-sedation when stacked(moderate)
  • SSRIs: no documented adverse interaction; co-administration described in Russian protocols(minor)

Which Should You Take?

DHEA comes out ahead for most readers on the criteria we weight: 2 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-A outcome catalogued. Selank is the right call when one of the conditionals below applies.

  • If your priority is hormonal optimization, pick DHEA.
  • If your priority is healthspan extension, pick DHEA.
  • If your priority is focus or working memory, pick Selank.
  • If your priority is anxiety reduction, pick Selank.

Edge case: If you want to avoid research-only / gray-market sourcing, DHEA is the more accessible choice.

Default choice: DHEA. Lower friction to source, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for Selank only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between DHEA and Selank?

DHEA and Selank differ in category (hormone vs peptide), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, DHEA or Selank?

DHEA half-life is 12 hours; Selank half-life is 0.5 hours.

Can you stack DHEA with Selank?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

Go deeper