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Comparison

EGCG vs Semax

Side-by-side of EGCG and Semax. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

EGCG

  • Modest fat loss (~1.3 kg over 12 weeks) when combined with caffeine and caloric deficit
  • Small reductions in LDL cholesterol (3-6 mg/dL) and systolic blood pressure (2-3 mmHg)
  • EFSA flags hepatotoxicity risk above 800 mg/day, particularly when taken fasted
  • Bioavailability is 0.1-1.0%; gut microbiome variation drives population-variable response
  • Green tea extract typically combines EGCG with caffeine and L-theanine for additive effects
  • Reduces non-heme iron absorption when co-administered with meals

Semax

  • Synthetic heptapeptide analog of ACTH(4-10) developed in Russia in the 1980s
  • Approved in Russia for ischemic stroke, cognitive impairment, and cerebrovascular disorders
  • Lacks the corticotropic activity of native ACTH due to the Pro-Gly-Pro stabilizing tail
  • Russian RCTs report improved cognitive recovery in acute ischemic stroke versus standard care
  • Modulates BDNF and NGF expression and dopaminergic signaling in preclinical models
  • Standard route is intranasal; not FDA approved; research-use-only outside Russia

Side-by-side

Attribute EGCG Semax
Category natural peptide
Also known as epigallocatechin gallate, green tea extract Met-Glu-His-Phe-Pro-Gly-Pro, ACTH(4-10) Pro-Gly-Pro analog
Half-life (hr) 3 0.5
Typical dose (mg) 400 0.6
Dosing frequency 1 to 2 times daily with food 2-3x daily (intranasal)
Routes oral intranasal
Onset (hr) 1.5 0.5
Peak (hr) 2 2
Molecular weight 458.37 813.94
Molecular formula C22H18O11 C37H51N9O10S
Mechanism Inhibits catechol-O-methyltransferase (COMT) to prolong norepinephrine signaling; activates AMPK; scavenges reactive oxygen species via gallate ester; modulates gut microbiome and pancreatic lipase activity. Modulates BDNF and NGF expression in hippocampus and cortex, enhances dopaminergic and serotonergic signaling, and reduces oxidative stress markers in preclinical ischemia models. Lacks corticotropic activity of native ACTH.
Legal status Dietary supplement; warning labels required above 800 mg/day in some EU jurisdictions Approved in Russia for stroke and cognitive disorders; not FDA approved; research-use-only grey market elsewhere
WADA status allowed unknown
DEA / Rx Not scheduled Not FDA approved; not scheduled; research-chemical status outside Russia
Pregnancy Avoid high-dose extracts; moderate green tea consumption appears acceptable Not recommended; insufficient data
CAS 989-51-5 80714-61-0
PubChem CID 65064 9811102
Wikidata Q307091 Q4413083

Safety profile

EGCG

Common side effects

  • nausea
  • abdominal discomfort
  • diarrhea
  • jitteriness (with caffeine)
  • sleep disruption (with caffeine)

Contraindications

  • pregnancy at high-dose extracts
  • active liver disease
  • iron deficiency anemia (separate dosing)

Interactions

  • iron supplements: reduces non-heme iron absorption; separate by 2 to 3 hours(moderate)
  • anticoagulants: additive effects at high catechin doses(minor)
  • beta-blockers (nadolol): reduced absorption when taken simultaneously(moderate)
  • hepatotoxic supplements (high-dose niacin, kava): theoretical additive hepatotoxicity at high EGCG doses(moderate)
  • stimulants and caffeine: additive thermogenic and cardiovascular effects(minor)

Semax

Common side effects

  • mild nasal irritation
  • transient mild headache
  • rare mild euphoria or activation

Contraindications

  • pregnancy
  • lactation
  • acute psychotic disorder
  • severe hypertension (caution due to mild activating effect)

Interactions

  • stimulants (caffeine, amphetamines): potential additive activation; monitor for overstimulation(minor)
  • antipsychotics: theoretical antagonism via dopaminergic modulation(minor)

Which Should You Take?

EGCG comes out ahead for most readers on the criteria we weight: 3 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-B outcome catalogued. Semax is the right call when one of the conditionals below applies.

  • If your priority is metabolic health and glucose control, pick EGCG.
  • If your priority is healthspan extension, pick EGCG.
  • If your priority is focus or working memory, pick Semax.
  • If your priority is long-term neuroprotection, pick Semax.

Edge case: If you want to avoid research-only / gray-market sourcing, EGCG is the more accessible choice.

Default choice: EGCG. Lower friction to source, and broader goal coverage. Reach for Semax only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between EGCG and Semax?

EGCG and Semax differ in category (natural vs peptide), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, EGCG or Semax?

EGCG half-life is 3 hours; Semax half-life is 0.5 hours.

Can you stack EGCG with Semax?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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