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BiologicalX

Comparison

Fisetin vs Rapamycin

Side-by-side of Fisetin and Rapamycin. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Fisetin

  • Flavonoid found in strawberries; most potent natural senolytic in screening assays (Yousefzadeh 2018)
  • Hickson 2019 confirmed reduced senescent-cell burden in human adipose tissue at 20 mg/kg pulsed for 2 days
  • Pulsed Mayo protocol (20 mg/kg/day x 2 days monthly) is the only dose with human biomarker evidence
  • Daily low-dose (100-500 mg) is mechanistically weaker but commonly used
  • Low oral bioavailability; with-fat dosing modestly improves absorption
  • Active cancer is a relative contraindication pending clearer polyphenol-treatment data

Rapamycin

  • Inhibits mTORC1 signaling by binding FKBP12, reducing protein synthesis and relieving autophagy suppression
  • ITP mouse program reproduced lifespan extension of ~10 to 25% across multiple genetic backgrounds and sexes
  • Mannick trials showed improved influenza vaccine response in elderly adults using analogs of rapamycin
  • PEARL human trial reported acceptable safety at 5 to 10 mg weekly with some functional and lean-mass signals
  • Common dose-limiting adverse effects include stomatitis, acne-like rash, and mildly elevated lipid markers
  • CYP3A4 substrate: grapefruit, ketoconazole, and clarithromycin substantially raise rapamycin exposure

Side-by-side

Attribute Fisetin Rapamycin
Category supplement pharmaceutical
Also known as 3,7,3',4'-tetrahydroxyflavone Sirolimus, Rapamune
Half-life (hr) 2 62
Typical dose (mg) 500 6
Dosing frequency pulsed 2 days/month (Mayo protocol) or daily continuous (empirical) weekly (longevity protocols); daily for transplant indication
Routes oral oral
Onset (hr) 1 1
Peak (hr) 4 2
Molecular weight 286.24 914.17
Molecular formula C15H10O6 C51H79NO13
Mechanism Senolytic via Bcl-2 family inhibition (Bcl-xL, Bcl-w); broad polyphenol with Nrf2 activation, mTOR inhibition at high concentrations, and antioxidant effects. Binds FKBP12, and the resulting complex inhibits mTORC1, reducing protein synthesis and autophagy suppression downstream of nutrient and growth-factor signaling.
Legal status OTC dietary supplement Prescription only (off-label for longevity)
WADA status allowed allowed
DEA / Rx OTC supplement Rx only (not a controlled substance)
Pregnancy Insufficient data Not recommended
CAS 528-48-3 53123-88-9
PubChem CID 5281614 5284616
Wikidata Q230614 Q410174

Safety profile

Fisetin

Common side effects

  • mild GI upset
  • headache (rare)

Contraindications

  • active cancer (theoretical, polyphenol interactions)
  • pregnancy and lactation (insufficient data)
  • concurrent CYP3A4-sensitive medications

Interactions

  • statins (CYP3A4 substrates): theoretical reduction in statin clearance at high fisetin doses(minor)
  • warfarin: theoretical CYP-mediated interaction; monitor INR if combining(moderate)
  • other senolytics (rapamycin, dasatinib + quercetin): additive senolytic effect; pairing is investigational(minor)

Rapamycin

Common side effects

  • mouth ulcers (stomatitis)
  • acne-like rash
  • GI upset
  • altered lipid panel
  • delayed wound healing

Contraindications

  • active infection
  • severe hepatic impairment
  • planned surgery (delayed wound healing)
  • pregnancy
  • live vaccines within dosing window

Interactions

  • strong CYP3A4 inhibitors (ketoconazole, clarithromycin, grapefruit): substantially raises rapamycin levels, toxicity risk(major)
  • strong CYP3A4 inducers (rifampin, St John's wort): lowers rapamycin levels, reduced effect(major)
  • ACE inhibitors: increased risk of angioedema(moderate)
  • live vaccines: reduced vaccine efficacy due to immunosuppression(major)

Which Should You Take?

Fisetin comes out ahead for most readers on the criteria we weight: 2 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-B outcome catalogued. Rapamycin is the right call when one of the conditionals below applies.

  • If your priority is focus or working memory, pick Fisetin.
  • If your priority is immune support, pick Rapamycin.
  • If your priority is healthspan extension, pick Fisetin.

Edge case: If you want to avoid prescription-only, Fisetin is the more accessible choice.

Default choice: Fisetin. Lower friction to source, and broader goal coverage. Reach for Rapamycin only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Fisetin and Rapamycin?

Fisetin and Rapamycin differ in category (supplement vs pharmaceutical), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Fisetin or Rapamycin?

Fisetin half-life is 2 hours; Rapamycin half-life is 62 hours.

Can you stack Fisetin with Rapamycin?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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