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Comparison

Fisetin vs Vitamin D3 + K2

Side-by-side of Fisetin and Vitamin D3 + K2. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Fisetin

  • Flavonoid found in strawberries; most potent natural senolytic in screening assays (Yousefzadeh 2018)
  • Hickson 2019 confirmed reduced senescent-cell burden in human adipose tissue at 20 mg/kg pulsed for 2 days
  • Pulsed Mayo protocol (20 mg/kg/day x 2 days monthly) is the only dose with human biomarker evidence
  • Daily low-dose (100-500 mg) is mechanistically weaker but commonly used
  • Low oral bioavailability; with-fat dosing modestly improves absorption
  • Active cancer is a relative contraindication pending clearer polyphenol-treatment data

Vitamin D3 + K2

  • Reduces non-vertebral fractures 10-20% in older adults at 800 IU/day or above when combined with calcium
  • VITAL trial showed neutral results on primary CV and cancer endpoints at 2000 IU/day over 5 years
  • Vitamin D supplementation reduces respiratory infection incidence ~10-20% in deficient populations
  • K2 MK-7 has 72-hour plasma half-life vs 1-2 hours for MK-4; once-daily dosing is sufficient
  • Synergy hypothesis is largely preclinical; dedicated combination RCTs are limited
  • Daily dosing outperforms bolus dosing for immune and infection outcomes

Side-by-side

Attribute Fisetin Vitamin D3 + K2
Category supplement supplement
Also known as 3,7,3',4'-tetrahydroxyflavone cholecalciferol + menaquinone, D3/K2, vitamin D3 with MK-7
Half-life (hr) 2 360
Typical dose (mg) 500 0.05
Dosing frequency pulsed 2 days/month (Mayo protocol) or daily continuous (empirical) daily with a fat-containing meal
Routes oral oral
Onset (hr) 1 24
Peak (hr) 4 168
Molecular weight 286.24 384.64
Molecular formula C15H10O6 C27H44O (D3); C46H64O2 (MK-7)
Mechanism Senolytic via Bcl-2 family inhibition (Bcl-xL, Bcl-w); broad polyphenol with Nrf2 activation, mTOR inhibition at high concentrations, and antioxidant effects. D3 converts to calcidiol then calcitriol, activating the vitamin D receptor (VDR) to increase intestinal calcium absorption and modulate immune and bone gene transcription. K2 carboxylates osteocalcin and matrix Gla protein, directing calcium toward bone and inhibiting vascular calcification.
Legal status OTC dietary supplement Dietary supplement (global)
WADA status allowed allowed
DEA / Rx OTC supplement Not scheduled
Pregnancy Insufficient data Recommended at standard doses for fetal bone development; consult clinician at higher doses
CAS 528-48-3 67-97-0
PubChem CID 5281614 5280795
Wikidata Q230614 Q139347

Safety profile

Fisetin

Common side effects

  • mild GI upset
  • headache (rare)

Contraindications

  • active cancer (theoretical, polyphenol interactions)
  • pregnancy and lactation (insufficient data)
  • concurrent CYP3A4-sensitive medications

Interactions

  • statins (CYP3A4 substrates): theoretical reduction in statin clearance at high fisetin doses(minor)
  • warfarin: theoretical CYP-mediated interaction; monitor INR if combining(moderate)
  • other senolytics (rapamycin, dasatinib + quercetin): additive senolytic effect; pairing is investigational(minor)

Vitamin D3 + K2

Common side effects

  • GI upset at high doses
  • headache (rare)
  • hypercalcemia (only at sustained very high D3 doses)

Contraindications

  • hypercalcemia
  • sarcoidosis
  • active hyperparathyroidism
  • warfarin therapy (K2 component requires stable intake)

Interactions

  • warfarin: K2 component can affect anticoagulation; maintain stable intake and inform anticoagulation clinic(moderate)
  • thiazide diuretics: additive calcium retention; hypercalcemia risk with high-dose D3(moderate)
  • digoxin and calcium channel blockers: additive effects from D3-induced hypercalcemia(moderate)
  • glucocorticoids: reduced vitamin D efficacy and bone effects(moderate)
  • cholestyramine and orlistat: bind fat-soluble vitamins; separate dosing by 2 to 4 hours(moderate)

Which Should You Take?

Vitamin D3 + K2 comes out ahead for most readers on the criteria we weight: 3 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-A outcome catalogued. Fisetin is the right call when one of the conditionals below applies.

  • If your priority is focus or working memory, pick Fisetin.
  • If your priority is bone density, pick Vitamin D3 + K2.
  • If your priority is cardiovascular health, pick Vitamin D3 + K2.

Edge case: Half-lives differ materially (Fisetin ~2 hr vs Vitamin D3 + K2 ~360 hr). Vitamin D3 + K2 reaches steady state faster; Fisetin is easier to dial in if tolerability is uncertain.

Default choice: Vitamin D3 + K2. Lower friction to source, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for Fisetin only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Fisetin and Vitamin D3 + K2?

Fisetin and Vitamin D3 + K2 differ in category (supplement vs supplement), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Fisetin or Vitamin D3 + K2?

Fisetin half-life is 2 hours; Vitamin D3 + K2 half-life is 360 hours.

Can you stack Fisetin with Vitamin D3 + K2?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

Go deeper