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Comparison

GHRP-2 vs Nicotinamide Riboside

Side-by-side of GHRP-2 and Nicotinamide Riboside. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

GHRP-2

  • Hexapeptide ghrelin-receptor agonist that stimulates pulsatile GH release within 15 to 30 minutes
  • Strongest appetite signal among GHRPs at standard doses; centrally mediated via NPY/AgRP
  • Produces measurable cortisol and prolactin rise (more than ipamorelin, less than GHRP-6)
  • Approved in Japan as pralmorelin for GH-deficiency diagnostic provocation; not FDA approved
  • Anecdotal protocols use 100 to 300 mcg subcutaneously 2 to 3 times daily on an empty stomach
  • Banned by WADA under S2; detection methods validated in accredited labs

Nicotinamide Riboside

  • Most-studied NAD+ precursor in human trials; the original Niagen formulation by Chromadex
  • Plasma NAD+ rises 30-60% at 250-1,000 mg/day across multiple human PK trials
  • Martens 2018 reported reduced BP and arterial stiffness at 500 mg/day for 6 weeks
  • Dollerup 2018 found no insulin sensitivity change despite plasma NAD+ rise
  • Tissue NAD+ rise inconsistent; hard clinical endpoints not yet measured
  • Larger human safety database than NMN; comparable mechanistic effects

Side-by-side

Attribute GHRP-2 Nicotinamide Riboside
Category peptide supplement
Also known as Growth Hormone Releasing Peptide 2, Pralmorelin, KP-102, GPA-748 NR, Niagen, nicotinamide riboside chloride
Half-life (hr) 0.5 8
Typical dose (mg) 0.1 500
Dosing frequency 2-3x daily daily, typically morning
Routes subcutaneous, intranasal, intravenous oral
Onset (hr) 0.25 1
Peak (hr) 0.5 4
Molecular weight 817.97 255.25
Molecular formula C45H55N9O6 C11H15N2O5
Mechanism Hexapeptide agonist of the growth-hormone secretagogue receptor (GHS-R1a). Suppresses hypothalamic somatostatin tone and stimulates pituitary somatotrophs, producing a pulsatile GH release with secondary cortisol, prolactin, and ACTH elevation. NAD+ precursor via salvage pathway. Phosphorylated to NMN by nicotinamide riboside kinase (NRK), then converted to NAD+. Substrate for sirtuins, PARPs, and CD38.
Legal status Not FDA approved; approved in Japan as pralmorelin (diagnostic); research-use-only grey market in US/EU; banned by WADA OTC dietary supplement
WADA status banned allowed
DEA / Rx Not scheduled in US (research chemical); approved diagnostic in Japan OTC supplement
Pregnancy Insufficient data; not recommended Insufficient data at supplement doses
CAS 158861-67-7 1341-23-7
PubChem CID 9919072 439924
Wikidata Q7235681 Q3343054

Safety profile

GHRP-2

Common side effects

  • acute hunger
  • head pressure or flushing
  • water retention
  • vivid dreams
  • tingling at injection site
  • transient lethargy

Contraindications

  • pregnancy
  • active malignancy
  • history of pituitary tumor
  • uncontrolled diabetes
  • severe insulin resistance

Interactions

  • CJC-1295: synergistic GH release; commonly co-administered for larger pulse(minor)
  • sermorelin: additive GH release via parallel GHRH and ghrelin pathways(minor)
  • insulin: sustained GH can blunt insulin sensitivity over weeks(moderate)
  • corticosteroids: blunt GH response and amplify cortisol load(moderate)

Nicotinamide Riboside

Common side effects

  • mild GI upset (rare)
  • headache (rare)

Contraindications

  • pregnancy / lactation (insufficient data)
  • active cancer (theoretical, no contraindicating data)

Interactions

  • pterostilbene: complementary sirtuin pathway (Basis combination)(minor)
  • TMG (trimethylglycine): methylation support during high NAD+ precursor dosing(minor)

Which Should You Take?

Nicotinamide Riboside comes out ahead for most readers on the criteria we weight: 3 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-A outcome catalogued. GHRP-2 is the right call when one of the conditionals below applies.

Edge case: If you want to avoid research-only / gray-market sourcing, Nicotinamide Riboside is the more accessible choice.

Default choice: Nicotinamide Riboside. Lower friction to source, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for GHRP-2 only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between GHRP-2 and Nicotinamide Riboside?

GHRP-2 and Nicotinamide Riboside differ in category (peptide vs supplement), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, GHRP-2 or Nicotinamide Riboside?

GHRP-2 half-life is 0.5 hours; Nicotinamide Riboside half-life is 8 hours.

Can you stack GHRP-2 with Nicotinamide Riboside?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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