Skip to content
BiologicalX

Comparison

GHRP-2 vs Vitamin D3 + K2

Side-by-side of GHRP-2 and Vitamin D3 + K2. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

GHRP-2

  • Hexapeptide ghrelin-receptor agonist that stimulates pulsatile GH release within 15 to 30 minutes
  • Strongest appetite signal among GHRPs at standard doses; centrally mediated via NPY/AgRP
  • Produces measurable cortisol and prolactin rise (more than ipamorelin, less than GHRP-6)
  • Approved in Japan as pralmorelin for GH-deficiency diagnostic provocation; not FDA approved
  • Anecdotal protocols use 100 to 300 mcg subcutaneously 2 to 3 times daily on an empty stomach
  • Banned by WADA under S2; detection methods validated in accredited labs

Vitamin D3 + K2

  • Reduces non-vertebral fractures 10-20% in older adults at 800 IU/day or above when combined with calcium
  • VITAL trial showed neutral results on primary CV and cancer endpoints at 2000 IU/day over 5 years
  • Vitamin D supplementation reduces respiratory infection incidence ~10-20% in deficient populations
  • K2 MK-7 has 72-hour plasma half-life vs 1-2 hours for MK-4; once-daily dosing is sufficient
  • Synergy hypothesis is largely preclinical; dedicated combination RCTs are limited
  • Daily dosing outperforms bolus dosing for immune and infection outcomes

Side-by-side

Attribute GHRP-2 Vitamin D3 + K2
Category peptide supplement
Also known as Growth Hormone Releasing Peptide 2, Pralmorelin, KP-102, GPA-748 cholecalciferol + menaquinone, D3/K2, vitamin D3 with MK-7
Half-life (hr) 0.5 360
Typical dose (mg) 0.1 0.05
Dosing frequency 2-3x daily daily with a fat-containing meal
Routes subcutaneous, intranasal, intravenous oral
Onset (hr) 0.25 24
Peak (hr) 0.5 168
Molecular weight 817.97 384.64
Molecular formula C45H55N9O6 C27H44O (D3); C46H64O2 (MK-7)
Mechanism Hexapeptide agonist of the growth-hormone secretagogue receptor (GHS-R1a). Suppresses hypothalamic somatostatin tone and stimulates pituitary somatotrophs, producing a pulsatile GH release with secondary cortisol, prolactin, and ACTH elevation. D3 converts to calcidiol then calcitriol, activating the vitamin D receptor (VDR) to increase intestinal calcium absorption and modulate immune and bone gene transcription. K2 carboxylates osteocalcin and matrix Gla protein, directing calcium toward bone and inhibiting vascular calcification.
Legal status Not FDA approved; approved in Japan as pralmorelin (diagnostic); research-use-only grey market in US/EU; banned by WADA Dietary supplement (global)
WADA status banned allowed
DEA / Rx Not scheduled in US (research chemical); approved diagnostic in Japan Not scheduled
Pregnancy Insufficient data; not recommended Recommended at standard doses for fetal bone development; consult clinician at higher doses
CAS 158861-67-7 67-97-0
PubChem CID 9919072 5280795
Wikidata Q7235681 Q139347

Safety profile

GHRP-2

Common side effects

  • acute hunger
  • head pressure or flushing
  • water retention
  • vivid dreams
  • tingling at injection site
  • transient lethargy

Contraindications

  • pregnancy
  • active malignancy
  • history of pituitary tumor
  • uncontrolled diabetes
  • severe insulin resistance

Interactions

  • CJC-1295: synergistic GH release; commonly co-administered for larger pulse(minor)
  • sermorelin: additive GH release via parallel GHRH and ghrelin pathways(minor)
  • insulin: sustained GH can blunt insulin sensitivity over weeks(moderate)
  • corticosteroids: blunt GH response and amplify cortisol load(moderate)

Vitamin D3 + K2

Common side effects

  • GI upset at high doses
  • headache (rare)
  • hypercalcemia (only at sustained very high D3 doses)

Contraindications

  • hypercalcemia
  • sarcoidosis
  • active hyperparathyroidism
  • warfarin therapy (K2 component requires stable intake)

Interactions

  • warfarin: K2 component can affect anticoagulation; maintain stable intake and inform anticoagulation clinic(moderate)
  • thiazide diuretics: additive calcium retention; hypercalcemia risk with high-dose D3(moderate)
  • digoxin and calcium channel blockers: additive effects from D3-induced hypercalcemia(moderate)
  • glucocorticoids: reduced vitamin D efficacy and bone effects(moderate)
  • cholestyramine and orlistat: bind fat-soluble vitamins; separate dosing by 2 to 4 hours(moderate)

Which Should You Take?

Vitamin D3 + K2 comes out ahead for most readers on the criteria we weight: 3 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-A outcome catalogued. GHRP-2 is the right call when one of the conditionals below applies.

  • If your priority is growth-hormone axis, pick GHRP-2.
  • If your priority is post-training recovery, pick GHRP-2.
  • If your priority is bone density, pick Vitamin D3 + K2.
  • If your priority is healthspan extension, pick Vitamin D3 + K2.

Edge case: If you want to avoid research-only / gray-market sourcing, Vitamin D3 + K2 is the more accessible choice.

Default choice: Vitamin D3 + K2. Lower friction to source, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for GHRP-2 only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between GHRP-2 and Vitamin D3 + K2?

GHRP-2 and Vitamin D3 + K2 differ in category (peptide vs supplement), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, GHRP-2 or Vitamin D3 + K2?

GHRP-2 half-life is 0.5 hours; Vitamin D3 + K2 half-life is 360 hours.

Can you stack GHRP-2 with Vitamin D3 + K2?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

Go deeper