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Comparison

Ipamorelin vs Low-Dose Naltrexone

Side-by-side of Ipamorelin and Low-Dose Naltrexone. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Ipamorelin

  • Pentapeptide GHS-R1a agonist with the cleanest selectivity profile in the GHRP class
  • Minimal cortisol and prolactin elevation at standard doses (substantially less than GHRP-2 or hexarelin)
  • ~2 hour plasma half-life, longest of the synthetic GHRPs
  • Largest human safety database (~600 participants in Helsinn's postoperative ileus phase 2)
  • Standard pairing for CJC-1295 no-DAC at 200 to 300 mcg subcutaneously 2 to 3 times daily
  • Banned by WADA under S2; never reached registration despite phase 2b development

Low-Dose Naltrexone

  • Off-label use at 1.5 to 4.5 mg, roughly one-tenth the FDA-approved 50 mg addiction-treatment dose
  • Proposed mechanisms include brief opioid receptor blockade triggering rebound endogenous opioid release, plus TLR4 antagonism
  • Compounded prescription only; insurance rarely covers; cash prices 20 to 80 USD per month
  • Younger 2013 reported ~30% pain reduction in fibromyalgia at 4.5 mg in a small crossover trial
  • Smith 2011 reported endoscopic improvement in active Crohn's disease (n=40 placebo-controlled)
  • Vivid dreams affect 20 to 40% in first 2 weeks; manageable by switching to morning dosing

Side-by-side

Attribute Ipamorelin Low-Dose Naltrexone
Category peptide pharmaceutical
Also known as NNC 26-0161, Aib-His-D-2-Nal-D-Phe-Lys-NH2 LDN, naltrexone (low dose)
Half-life (hr) 2 4
Typical dose (mg) 0.2 4.5
Dosing frequency 2-3x daily once daily, typically at bedtime
Routes subcutaneous, intravenous oral
Onset (hr) 0.25 1
Peak (hr) 1 1.5
Molecular weight 711.86 341.4
Molecular formula C38H49N9O5 C20H23NO4
Mechanism Selective GHS-R1a agonist that stimulates pulsatile GH release with minimal cortisol or prolactin co-activation. Suppresses hypothalamic somatostatin and stimulates pituitary somatotrophs. Brief mu-opioid receptor antagonism proposed to trigger compensatory upregulation of endogenous opioids; secondary TLR4 antagonism on microglia and immune cells contributes to anti-inflammatory effect.
Legal status Not FDA approved; advanced through phase 2b in postoperative ileus before discontinuation; research-use-only grey market; banned by WADA Off-label compounded prescription (naltrexone is FDA approved for opioid and alcohol use disorder at 50 mg)
WADA status banned allowed
DEA / Rx Not scheduled (research chemical) Rx only (not a controlled substance)
Pregnancy Insufficient data; not recommended Insufficient data; not routinely recommended
CAS 170851-70-4 16590-41-3
PubChem CID 11338566 5360515
Wikidata Q1666741 Q426444

Safety profile

Ipamorelin

Common side effects

  • injection-site irritation
  • vivid dreams
  • transient mild head pressure
  • occasional headache

Contraindications

  • pregnancy
  • active malignancy
  • history of pituitary tumor
  • uncontrolled diabetes

Interactions

  • CJC-1295: synergistic GH release via parallel GHRH and ghrelin pathways; standard pairing(minor)
  • sermorelin: additive GH release; functionally similar pairing to CJC-1295 with shorter GHRH half-life(minor)
  • insulin: sustained GH can blunt insulin sensitivity over weeks(moderate)
  • corticosteroids: blunt GH response; reduce expected efficacy(moderate)

Low-Dose Naltrexone

Common side effects

  • vivid dreams
  • sleep disruption
  • headache
  • mild GI upset
  • fatigue (early)

Contraindications

  • concurrent opioid use
  • acute hepatitis or liver failure
  • opioid dependence
  • pregnancy (insufficient data)

Interactions

  • opioid analgesics (oxycodone, morphine, codeine): blocks analgesic effect; precipitates withdrawal in dependent users(major)
  • tramadol: blocks opioid component of analgesia(major)
  • thyroid hormone replacement: may alter dose requirements after immune modulation; monitor TSH(minor)

Which Should You Take?

Low-Dose Naltrexone comes out ahead for most readers on the criteria we weight: 2 catalogued goals, prescription-only, oral dosing, with a Tier-A outcome catalogued. Ipamorelin is the right call when one of the conditionals below applies.

Edge case: If you cannot self-administer injections, Low-Dose Naltrexone is the only oral option in this pair.

Default choice: Low-Dose Naltrexone. Wider use case, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for Ipamorelin only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Ipamorelin and Low-Dose Naltrexone?

Ipamorelin and Low-Dose Naltrexone differ in category (peptide vs pharmaceutical), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Ipamorelin or Low-Dose Naltrexone?

Ipamorelin half-life is 2 hours; Low-Dose Naltrexone half-life is 4 hours.

Can you stack Ipamorelin with Low-Dose Naltrexone?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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