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BiologicalX

Comparison

Ipamorelin vs Modafinil

Side-by-side of Ipamorelin and Modafinil. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Ipamorelin

  • Pentapeptide GHS-R1a agonist with the cleanest selectivity profile in the GHRP class
  • Minimal cortisol and prolactin elevation at standard doses (substantially less than GHRP-2 or hexarelin)
  • ~2 hour plasma half-life, longest of the synthetic GHRPs
  • Largest human safety database (~600 participants in Helsinn's postoperative ileus phase 2)
  • Standard pairing for CJC-1295 no-DAC at 200 to 300 mcg subcutaneously 2 to 3 times daily
  • Banned by WADA under S2; never reached registration despite phase 2b development

Modafinil

  • FDA approved in 1998 for narcolepsy, with later additions for shift-work sleep disorder and OSA residual sleepiness
  • Schedule IV controlled substance in the US; prescription-only in EU, UK, Australia
  • Increases wakefulness via weak dopamine reuptake inhibition plus histaminergic, noradrenergic, and orexinergic activation
  • Long half-life of 12 to 15 hours requires morning dosing to avoid sleep disruption
  • Modest cognitive enhancement signal in non-sleep-deprived adults at 100 to 200 mg (Battleday meta-review 2015)
  • Substantial CYP3A4 induction reduces hormonal contraceptive efficacy; barrier methods recommended

Side-by-side

Attribute Ipamorelin Modafinil
Category peptide pharmaceutical
Also known as NNC 26-0161, Aib-His-D-2-Nal-D-Phe-Lys-NH2 Provigil, Modalert, Modvigil, diphenylmethylsulfinyl-acetamide
Half-life (hr) 2 13
Typical dose (mg) 0.2 200
Dosing frequency 2-3x daily daily, morning
Routes subcutaneous, intravenous oral
Onset (hr) 0.25 1
Peak (hr) 1 3
Molecular weight 711.86 273.35
Molecular formula C38H49N9O5 C15H15NO2S
Mechanism Selective GHS-R1a agonist that stimulates pulsatile GH release with minimal cortisol or prolactin co-activation. Suppresses hypothalamic somatostatin and stimulates pituitary somatotrophs. Weak dopamine reuptake inhibition plus downstream activation of histaminergic, noradrenergic, and orexinergic wake-promoting systems.
Legal status Not FDA approved; advanced through phase 2b in postoperative ileus before discontinuation; research-use-only grey market; banned by WADA Schedule IV (US); prescription-only globally; not a supplement
WADA status banned banned
DEA / Rx Not scheduled (research chemical) Schedule IV
Pregnancy Insufficient data; not recommended Not recommended
CAS 170851-70-4 68693-11-8
PubChem CID 11338566 4236
Wikidata Q1666741 Q422968

Safety profile

Ipamorelin

Common side effects

  • injection-site irritation
  • vivid dreams
  • transient mild head pressure
  • occasional headache

Contraindications

  • pregnancy
  • active malignancy
  • history of pituitary tumor
  • uncontrolled diabetes

Interactions

  • CJC-1295: synergistic GH release via parallel GHRH and ghrelin pathways; standard pairing(minor)
  • sermorelin: additive GH release; functionally similar pairing to CJC-1295 with shorter GHRH half-life(minor)
  • insulin: sustained GH can blunt insulin sensitivity over weeks(moderate)
  • corticosteroids: blunt GH response; reduce expected efficacy(moderate)

Modafinil

Common side effects

  • headache
  • nausea
  • anxiety
  • insomnia (with late-day dosing)
  • dry mouth
  • mild blood pressure elevation

Contraindications

  • recent myocardial infarction
  • unstable angina
  • left ventricular hypertrophy
  • significant arrhythmia
  • history of Stevens-Johnson syndrome
  • psychotic disorders
  • pregnancy
  • concurrent MAOI use

Interactions

  • hormonal contraceptives: CYP3A4 induction reduces contraceptive efficacy; use barrier method(major)
  • cyclosporine: reduced cyclosporine levels via CYP3A4 induction(major)
  • warfarin: CYP2C9 inhibition raises INR(moderate)
  • phenytoin: CYP2C19 inhibition raises phenytoin levels(moderate)
  • MAOIs: potential hypertensive reaction(major)
  • classical stimulants (amphetamine, methylphenidate): additive cardiovascular and sleep-disruption effects(moderate)

Which Should You Take?

Modafinil comes out ahead for most readers on the criteria we weight: 3 catalogued goals, controlled substance, oral dosing, with a Tier-A outcome catalogued. Ipamorelin is the right call when one of the conditionals below applies.

  • If your priority is growth-hormone axis, pick Ipamorelin.
  • If your priority is post-training recovery, pick Ipamorelin.
  • If your priority is wakefulness, pick Modafinil.
  • If your priority is focus or working memory, pick Modafinil.

Edge case: If you cannot self-administer injections, Modafinil is the only oral option in this pair.

Default choice: Modafinil. Wider use case, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for Ipamorelin only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Ipamorelin and Modafinil?

Ipamorelin and Modafinil differ in category (peptide vs pharmaceutical), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Ipamorelin or Modafinil?

Ipamorelin half-life is 2 hours; Modafinil half-life is 13 hours.

Can you stack Ipamorelin with Modafinil?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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