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Comparison

Low-Dose Naltrexone vs Selank

Side-by-side of Low-Dose Naltrexone and Selank. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Low-Dose Naltrexone

  • Off-label use at 1.5 to 4.5 mg, roughly one-tenth the FDA-approved 50 mg addiction-treatment dose
  • Proposed mechanisms include brief opioid receptor blockade triggering rebound endogenous opioid release, plus TLR4 antagonism
  • Compounded prescription only; insurance rarely covers; cash prices 20 to 80 USD per month
  • Younger 2013 reported ~30% pain reduction in fibromyalgia at 4.5 mg in a small crossover trial
  • Smith 2011 reported endoscopic improvement in active Crohn's disease (n=40 placebo-controlled)
  • Vivid dreams affect 20 to 40% in first 2 weeks; manageable by switching to morning dosing

Selank

  • Synthetic heptapeptide analog of tuftsin developed in Russia in the 1990s
  • Approved in Russia for generalized anxiety disorder and asthenic conditions
  • Russian RCTs report anxiolytic effects comparable to medazepam without sedation or dependence
  • Modulates GABAergic and serotonergic signaling and BDNF expression in preclinical models
  • Most commonly administered intranasally; subcutaneous use is anecdotal
  • No Western-validated trials; not FDA approved; research-use-only outside Russia

Side-by-side

Attribute Low-Dose Naltrexone Selank
Category pharmaceutical peptide
Also known as LDN, naltrexone (low dose) TP-7, Tuftsin analog
Half-life (hr) 4 0.5
Typical dose (mg) 4.5 0.4
Dosing frequency once daily, typically at bedtime 2-3x daily (intranasal)
Routes oral intranasal, subcutaneous
Onset (hr) 1 0.25
Peak (hr) 1.5 1
Molecular weight 341.4 751.85
Molecular formula C20H23NO4 C33H57N11O9
Mechanism Brief mu-opioid receptor antagonism proposed to trigger compensatory upregulation of endogenous opioids; secondary TLR4 antagonism on microglia and immune cells contributes to anti-inflammatory effect. Modulates GABAergic, serotonergic, and dopaminergic signaling. Increases BDNF expression in hippocampal neurons in preclinical models. Modulates enkephalin levels and immune cytokine signaling via tuftsin-like activity.
Legal status Off-label compounded prescription (naltrexone is FDA approved for opioid and alcohol use disorder at 50 mg) Approved as a prescription anxiolytic in Russia; not FDA approved; research-use-only grey market in most other jurisdictions
WADA status allowed unknown
DEA / Rx Rx only (not a controlled substance) Not FDA approved; not scheduled; research-chemical status outside Russia
Pregnancy Insufficient data; not routinely recommended Not recommended; insufficient data
CAS 16590-41-3 129954-34-3
PubChem CID 5360515 11765600
Wikidata Q426444 Q4416793

Safety profile

Low-Dose Naltrexone

Common side effects

  • vivid dreams
  • sleep disruption
  • headache
  • mild GI upset
  • fatigue (early)

Contraindications

  • concurrent opioid use
  • acute hepatitis or liver failure
  • opioid dependence
  • pregnancy (insufficient data)

Interactions

  • opioid analgesics (oxycodone, morphine, codeine): blocks analgesic effect; precipitates withdrawal in dependent users(major)
  • tramadol: blocks opioid component of analgesia(major)
  • thyroid hormone replacement: may alter dose requirements after immune modulation; monitor TSH(minor)

Selank

Common side effects

  • mild nasal irritation (intranasal)
  • transient drowsiness (uncommon)
  • mild headache

Contraindications

  • pregnancy
  • lactation
  • severe psychiatric disorder (insufficient data)

Interactions

  • benzodiazepines: additive anxiolytic effect; potential for over-sedation when stacked(moderate)
  • SSRIs: no documented adverse interaction; co-administration described in Russian protocols(minor)

Which Should You Take?

Low-Dose Naltrexone comes out ahead for most readers on the criteria we weight: 2 catalogued goals, prescription-only, oral dosing, with a Tier-A outcome catalogued. Selank is the right call when one of the conditionals below applies.

  • If your priority is immune support, pick Low-Dose Naltrexone.
  • If your priority is pain modulation, pick Low-Dose Naltrexone.
  • If your priority is focus or working memory, pick Selank.
  • If your priority is anxiety reduction, pick Selank.

Edge case: If you cannot self-administer injections, Low-Dose Naltrexone is the only oral option in this pair.

Default choice: Low-Dose Naltrexone. Wider use case, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for Selank only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Low-Dose Naltrexone and Selank?

Low-Dose Naltrexone and Selank differ in category (pharmaceutical vs peptide), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Low-Dose Naltrexone or Selank?

Low-Dose Naltrexone half-life is 4 hours; Selank half-life is 0.5 hours.

Can you stack Low-Dose Naltrexone with Selank?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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