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BiologicalX

Comparison

Noopept vs Rapamycin

Side-by-side of Noopept and Rapamycin. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Noopept

  • Russian dipeptide nootropic developed in the 1990s, registered in Russia 2002 for cognitive impairment
  • Roughly 1,000-fold higher per-mg potency than piracetam; therapeutic dose 10 to 30 mg/day
  • Active metabolite cycloprolylglycine modulates AMPA receptors and increases NGF and BDNF in rodent hippocampus
  • Russian RCTs in stroke recovery and vascular cognitive impairment show modest improvements over 4 to 8 weeks
  • Western evidence base is essentially absent; healthy-adult enhancement trials have not been published
  • Unscheduled in the US but not approved for human consumption; UK is prescription-only since 2014

Rapamycin

  • Inhibits mTORC1 signaling by binding FKBP12, reducing protein synthesis and relieving autophagy suppression
  • ITP mouse program reproduced lifespan extension of ~10 to 25% across multiple genetic backgrounds and sexes
  • Mannick trials showed improved influenza vaccine response in elderly adults using analogs of rapamycin
  • PEARL human trial reported acceptable safety at 5 to 10 mg weekly with some functional and lean-mass signals
  • Common dose-limiting adverse effects include stomatitis, acne-like rash, and mildly elevated lipid markers
  • CYP3A4 substrate: grapefruit, ketoconazole, and clarithromycin substantially raise rapamycin exposure

Side-by-side

Attribute Noopept Rapamycin
Category nootropic pharmaceutical
Also known as GVS-111, N-phenylacetyl-L-prolylglycine ethyl ester, Omberacetam Sirolimus, Rapamune
Half-life (hr) 0.7 62
Typical dose (mg) 20 6
Dosing frequency 2 to 3 times daily, last dose before mid-afternoon weekly (longevity protocols); daily for transplant indication
Routes oral, sublingual oral
Onset (hr) 0.5 1
Peak (hr) 1 2
Molecular weight 318.37 914.17
Molecular formula C17H22N2O4 C51H79NO13
Mechanism Hydrolyzed to active metabolite cycloprolylglycine; AMPA receptor modulation, BDNF and NGF upregulation, antioxidant and antiexcitotoxic effects. Binds FKBP12, and the resulting complex inhibits mTORC1, reducing protein synthesis and autophagy suppression downstream of nutrient and growth-factor signaling.
Legal status Approved in Russia and CIS states; prescription-only in UK; unscheduled and unapproved in US, EU varies Prescription only (off-label for longevity)
WADA status unknown allowed
DEA / Rx Not scheduled in the US Rx only (not a controlled substance)
Pregnancy Not recommended Not recommended
CAS 157115-85-0 53123-88-9
PubChem CID 183503 5284616
Wikidata Q4321022 Q410174

Safety profile

Noopept

Common side effects

  • headache
  • irritability
  • sleep disturbance with late-day dosing
  • occasional blood pressure elevation

Contraindications

  • pregnancy
  • lactation
  • pediatric use
  • severe hepatic impairment
  • severe renal impairment

Interactions

  • memantine and other glutamatergic agents: theoretical AMPA-pathway interaction(minor)
  • antidepressants: theoretical effect via BDNF axis, undocumented(minor)
  • antihypertensives: occasional blood pressure elevation may require monitoring(minor)

Rapamycin

Common side effects

  • mouth ulcers (stomatitis)
  • acne-like rash
  • GI upset
  • altered lipid panel
  • delayed wound healing

Contraindications

  • active infection
  • severe hepatic impairment
  • planned surgery (delayed wound healing)
  • pregnancy
  • live vaccines within dosing window

Interactions

  • strong CYP3A4 inhibitors (ketoconazole, clarithromycin, grapefruit): substantially raises rapamycin levels, toxicity risk(major)
  • strong CYP3A4 inducers (rifampin, St John's wort): lowers rapamycin levels, reduced effect(major)
  • ACE inhibitors: increased risk of angioedema(moderate)
  • live vaccines: reduced vaccine efficacy due to immunosuppression(major)

Which Should You Take?

Rapamycin comes out ahead for most readers on the criteria we weight: 2 catalogued goals, prescription-only, oral dosing, with a Tier-A outcome catalogued. Noopept is the right call when one of the conditionals below applies.

  • If your priority is focus or working memory, pick Noopept.
  • If your priority is memory, pick Noopept.
  • If your priority is healthspan extension, pick Rapamycin.
  • If your priority is immune support, pick Rapamycin.

Edge case: Half-lives differ materially (Noopept ~0.7 hr vs Rapamycin ~62 hr). Rapamycin reaches steady state faster; Noopept is easier to dial in if tolerability is uncertain.

Default choice: Rapamycin. Wider use case, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for Noopept only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Noopept and Rapamycin?

Noopept and Rapamycin differ in category (nootropic vs pharmaceutical), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Noopept or Rapamycin?

Noopept half-life is 0.7 hours; Rapamycin half-life is 62 hours.

Can you stack Noopept with Rapamycin?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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