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BiologicalX

Comparison

Noopept vs Selank

Side-by-side of Noopept and Selank. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Noopept

  • Russian dipeptide nootropic developed in the 1990s, registered in Russia 2002 for cognitive impairment
  • Roughly 1,000-fold higher per-mg potency than piracetam; therapeutic dose 10 to 30 mg/day
  • Active metabolite cycloprolylglycine modulates AMPA receptors and increases NGF and BDNF in rodent hippocampus
  • Russian RCTs in stroke recovery and vascular cognitive impairment show modest improvements over 4 to 8 weeks
  • Western evidence base is essentially absent; healthy-adult enhancement trials have not been published
  • Unscheduled in the US but not approved for human consumption; UK is prescription-only since 2014

Selank

  • Synthetic heptapeptide analog of tuftsin developed in Russia in the 1990s
  • Approved in Russia for generalized anxiety disorder and asthenic conditions
  • Russian RCTs report anxiolytic effects comparable to medazepam without sedation or dependence
  • Modulates GABAergic and serotonergic signaling and BDNF expression in preclinical models
  • Most commonly administered intranasally; subcutaneous use is anecdotal
  • No Western-validated trials; not FDA approved; research-use-only outside Russia

Side-by-side

Attribute Noopept Selank
Category nootropic peptide
Also known as GVS-111, N-phenylacetyl-L-prolylglycine ethyl ester, Omberacetam TP-7, Tuftsin analog
Half-life (hr) 0.7 0.5
Typical dose (mg) 20 0.4
Dosing frequency 2 to 3 times daily, last dose before mid-afternoon 2-3x daily (intranasal)
Routes oral, sublingual intranasal, subcutaneous
Onset (hr) 0.5 0.25
Peak (hr) 1 1
Molecular weight 318.37 751.85
Molecular formula C17H22N2O4 C33H57N11O9
Mechanism Hydrolyzed to active metabolite cycloprolylglycine; AMPA receptor modulation, BDNF and NGF upregulation, antioxidant and antiexcitotoxic effects. Modulates GABAergic, serotonergic, and dopaminergic signaling. Increases BDNF expression in hippocampal neurons in preclinical models. Modulates enkephalin levels and immune cytokine signaling via tuftsin-like activity.
Legal status Approved in Russia and CIS states; prescription-only in UK; unscheduled and unapproved in US, EU varies Approved as a prescription anxiolytic in Russia; not FDA approved; research-use-only grey market in most other jurisdictions
WADA status unknown unknown
DEA / Rx Not scheduled in the US Not FDA approved; not scheduled; research-chemical status outside Russia
Pregnancy Not recommended Not recommended; insufficient data
CAS 157115-85-0 129954-34-3
PubChem CID 183503 11765600
Wikidata Q4321022 Q4416793

Safety profile

Noopept

Common side effects

  • headache
  • irritability
  • sleep disturbance with late-day dosing
  • occasional blood pressure elevation

Contraindications

  • pregnancy
  • lactation
  • pediatric use
  • severe hepatic impairment
  • severe renal impairment

Interactions

  • memantine and other glutamatergic agents: theoretical AMPA-pathway interaction(minor)
  • antidepressants: theoretical effect via BDNF axis, undocumented(minor)
  • antihypertensives: occasional blood pressure elevation may require monitoring(minor)

Selank

Common side effects

  • mild nasal irritation (intranasal)
  • transient drowsiness (uncommon)
  • mild headache

Contraindications

  • pregnancy
  • lactation
  • severe psychiatric disorder (insufficient data)

Interactions

  • benzodiazepines: additive anxiolytic effect; potential for over-sedation when stacked(moderate)
  • SSRIs: no documented adverse interaction; co-administration described in Russian protocols(minor)

Which Should You Take?

Noopept comes out ahead for most readers on the criteria we weight: 3 catalogued goals, controlled substance, oral dosing, with a Tier-B outcome catalogued. Selank is the right call when one of the conditionals below applies.

  • If your priority is memory, pick Noopept.
  • If your priority is stress and HPA-axis regulation, pick Noopept.
  • If your priority is anxiety reduction, pick Selank.
  • If your priority is mood, pick Selank.

Edge case: If you cannot self-administer injections, Noopept is the only oral option in this pair.

Default choice: Noopept. Wider use case, and broader goal coverage. Reach for Selank only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Noopept and Selank?

Noopept and Selank differ in category (nootropic vs peptide), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Noopept or Selank?

Noopept half-life is 0.7 hours; Selank half-life is 0.5 hours.

Can you stack Noopept with Selank?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

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