Skip to content
BiologicalX

Comparison

Noopept vs Urolithin A

Side-by-side of Noopept and Urolithin A. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Noopept

  • Russian dipeptide nootropic developed in the 1990s, registered in Russia 2002 for cognitive impairment
  • Roughly 1,000-fold higher per-mg potency than piracetam; therapeutic dose 10 to 30 mg/day
  • Active metabolite cycloprolylglycine modulates AMPA receptors and increases NGF and BDNF in rodent hippocampus
  • Russian RCTs in stroke recovery and vascular cognitive impairment show modest improvements over 4 to 8 weeks
  • Western evidence base is essentially absent; healthy-adult enhancement trials have not been published
  • Unscheduled in the US but not approved for human consumption; UK is prescription-only since 2014

Urolithin A

  • Gut-microbiome-derived metabolite of pomegranate and walnut ellagitannins
  • Roughly 40% of adults are 'urolithin producers' from dietary intake; ~60% are non-producers
  • Ryu 2016 (Nature Medicine) reported lifespan extension in C. elegans and muscle benefits in aged rodents
  • Andreux 2019 first-in-human trial (n=60) established safety and mitochondrial gene-expression upregulation
  • Singh 2022 (n=66, 4 months, 1000 mg/day) reported improved muscle endurance in older adults
  • Most human trial portfolio is Amazentis-funded; independent replication is thin

Side-by-side

Attribute Noopept Urolithin A
Category nootropic supplement
Also known as GVS-111, N-phenylacetyl-L-prolylglycine ethyl ester, Omberacetam UA, Mitopure, ellagitannin metabolite
Half-life (hr) 0.7 17
Typical dose (mg) 20 500
Dosing frequency 2 to 3 times daily, last dose before mid-afternoon daily, morning with food
Routes oral, sublingual oral
Onset (hr) 0.5 2
Peak (hr) 1 4
Molecular weight 318.37 228.2
Molecular formula C17H22N2O4 C13H8O4
Mechanism Hydrolyzed to active metabolite cycloprolylglycine; AMPA receptor modulation, BDNF and NGF upregulation, antioxidant and antiexcitotoxic effects. Induces mitophagy via potentiation of PINK1/Parkin signaling, leading to selective degradation of damaged mitochondria. Secondary anti-inflammatory effects via NF-kB modulation.
Legal status Approved in Russia and CIS states; prescription-only in UK; unscheduled and unapproved in US, EU varies OTC dietary supplement (US GRAS 2018; EFSA Novel Food 2021)
WADA status unknown allowed
DEA / Rx Not scheduled in the US OTC supplement (not scheduled)
Pregnancy Not recommended Insufficient data; not routinely recommended
CAS 157115-85-0 1143-70-0
PubChem CID 183503 5488186
Wikidata Q4321022 Q27101321

Safety profile

Noopept

Common side effects

  • headache
  • irritability
  • sleep disturbance with late-day dosing
  • occasional blood pressure elevation

Contraindications

  • pregnancy
  • lactation
  • pediatric use
  • severe hepatic impairment
  • severe renal impairment

Interactions

  • memantine and other glutamatergic agents: theoretical AMPA-pathway interaction(minor)
  • antidepressants: theoretical effect via BDNF axis, undocumented(minor)
  • antihypertensives: occasional blood pressure elevation may require monitoring(minor)

Urolithin A

Common side effects

  • mild GI upset (rare)
  • soft stools (rare)

Contraindications

  • pregnancy and lactation (insufficient data)
  • active chemotherapy (consult oncology)

Interactions

  • chemotherapy agents: theoretical interaction with mitochondrial-targeting agents; consult oncologist(moderate)

Which Should You Take?

Urolithin A comes out ahead for most readers on the criteria we weight: 3 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-A outcome catalogued. Noopept is the right call when one of the conditionals below applies.

  • If your priority is focus or working memory, pick Noopept.
  • If your priority is memory, pick Noopept.
  • If your priority is healthspan extension, pick Urolithin A.
  • If your priority is muscle hypertrophy, pick Urolithin A.

Edge case: If you want to avoid controlled substance, Urolithin A is the more accessible choice.

Default choice: Urolithin A. Lower friction to source, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for Noopept only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Noopept and Urolithin A?

Noopept and Urolithin A differ in category (nootropic vs supplement), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Noopept or Urolithin A?

Noopept half-life is 0.7 hours; Urolithin A half-life is 17 hours.

Can you stack Noopept with Urolithin A?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

Go deeper