Skip to content
BiologicalX

Comparison

Rapamycin vs Sermorelin

Side-by-side of Rapamycin and Sermorelin. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Rapamycin

  • Inhibits mTORC1 signaling by binding FKBP12, reducing protein synthesis and relieving autophagy suppression
  • ITP mouse program reproduced lifespan extension of ~10 to 25% across multiple genetic backgrounds and sexes
  • Mannick trials showed improved influenza vaccine response in elderly adults using analogs of rapamycin
  • PEARL human trial reported acceptable safety at 5 to 10 mg weekly with some functional and lean-mass signals
  • Common dose-limiting adverse effects include stomatitis, acne-like rash, and mildly elevated lipid markers
  • CYP3A4 substrate: grapefruit, ketoconazole, and clarithromycin substantially raise rapamycin exposure

Sermorelin

  • Synthetic 29-amino-acid GHRH fragment; FDA approved 1997 for pediatric GH deficiency as Geref
  • Voluntarily discontinued by Serono in 2008 for commercial reasons; not safety-related
  • Compounded by 503A/503B pharmacies for off-label adult anti-aging and body-composition use
  • Produces physiologic pulsatile GH release; ~10 to 20 minute plasma half-life
  • Standard anti-aging clinic protocol: 200 to 500 mcg subcutaneously pre-bed, often with ipamorelin
  • Banned by WADA under S2 (peptide hormones, growth factors)

Side-by-side

Attribute Rapamycin Sermorelin
Category pharmaceutical peptide
Also known as Sirolimus, Rapamune Sermorelin acetate, GRF 1-29, Geref, GHRH (1-29) NH2
Half-life (hr) 62 0.25
Typical dose (mg) 6 0.3
Dosing frequency weekly (longevity protocols); daily for transplant indication 1-2x daily
Routes oral subcutaneous
Onset (hr) 1 0.25
Peak (hr) 2 0.5
Molecular weight 914.17 3357.88
Molecular formula C51H79NO13 C149H246N44O42S
Mechanism Binds FKBP12, and the resulting complex inhibits mTORC1, reducing protein synthesis and autophagy suppression downstream of nutrient and growth-factor signaling. Synthetic 29-amino-acid GHRH fragment that binds the GHRH receptor on pituitary somatotrophs to stimulate endogenous pulsatile GH synthesis and release while preserving the GH-IGF-1 negative feedback loop.
Legal status Prescription only (off-label for longevity) FDA approved 1997 (Geref, pediatric GHD); voluntarily discontinued by Serono 2008; compounded by 503A/503B pharmacies for off-label adult use; banned by WADA
WADA status allowed banned
DEA / Rx Rx only (not a controlled substance) Rx only via compounding (no controlled-substance schedule)
Pregnancy Not recommended Category C (historical labeling); not recommended in pregnancy
CAS 53123-88-9 86168-78-7
PubChem CID 5284616 16129617
Wikidata Q410174 Q416620

Safety profile

Rapamycin

Common side effects

  • mouth ulcers (stomatitis)
  • acne-like rash
  • GI upset
  • altered lipid panel
  • delayed wound healing

Contraindications

  • active infection
  • severe hepatic impairment
  • planned surgery (delayed wound healing)
  • pregnancy
  • live vaccines within dosing window

Interactions

  • strong CYP3A4 inhibitors (ketoconazole, clarithromycin, grapefruit): substantially raises rapamycin levels, toxicity risk(major)
  • strong CYP3A4 inducers (rifampin, St John's wort): lowers rapamycin levels, reduced effect(major)
  • ACE inhibitors: increased risk of angioedema(moderate)
  • live vaccines: reduced vaccine efficacy due to immunosuppression(major)

Sermorelin

Common side effects

  • injection-site pain or irritation
  • transient flushing
  • headache
  • vivid dreams (pre-bed dosing)

Contraindications

  • pregnancy
  • active malignancy
  • history of pituitary tumor
  • diabetic retinopathy (theoretical)
  • untreated hypothyroidism

Interactions

  • ipamorelin: synergistic GH release via parallel GHRH and ghrelin pathways; standard anti-aging clinic pairing(minor)
  • CJC-1295: pharmacologically redundant (both GHRH-pathway); typically not stacked(minor)
  • insulin: sustained GH can blunt insulin sensitivity over weeks(moderate)
  • corticosteroids: blunt GH response; reduce expected efficacy(moderate)
  • levothyroxine (untreated hypothyroidism): untreated hypothyroidism blunts GH response; correct thyroid first(moderate)

Which Should You Take?

Rapamycin and Sermorelin score evenly on the criteria we weight (goal breadth, legal accessibility, evidence depth). The conditionals below should drive the decision more than any aggregate score.

  • If your priority is immune support, pick Rapamycin.
  • If your priority is growth-hormone axis, pick Sermorelin.
  • If your priority is post-training recovery, pick Sermorelin.

Edge case: If you cannot self-administer injections, Rapamycin is the only oral option in this pair.

Default choice: either is defensible. Rapamycin edges out on goal breadth + legal accessibility; Sermorelin is the right call if your priority sits in the goals listed above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Rapamycin and Sermorelin?

Rapamycin and Sermorelin differ in category (pharmaceutical vs peptide), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Rapamycin or Sermorelin?

Rapamycin half-life is 62 hours; Sermorelin half-life is 0.25 hours.

Can you stack Rapamycin with Sermorelin?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

Go deeper