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Comparison

Rapamycin vs Vitamin D3 + K2

Side-by-side of Rapamycin and Vitamin D3 + K2. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

Rapamycin

  • Inhibits mTORC1 signaling by binding FKBP12, reducing protein synthesis and relieving autophagy suppression
  • ITP mouse program reproduced lifespan extension of ~10 to 25% across multiple genetic backgrounds and sexes
  • Mannick trials showed improved influenza vaccine response in elderly adults using analogs of rapamycin
  • PEARL human trial reported acceptable safety at 5 to 10 mg weekly with some functional and lean-mass signals
  • Common dose-limiting adverse effects include stomatitis, acne-like rash, and mildly elevated lipid markers
  • CYP3A4 substrate: grapefruit, ketoconazole, and clarithromycin substantially raise rapamycin exposure

Vitamin D3 + K2

  • Reduces non-vertebral fractures 10-20% in older adults at 800 IU/day or above when combined with calcium
  • VITAL trial showed neutral results on primary CV and cancer endpoints at 2000 IU/day over 5 years
  • Vitamin D supplementation reduces respiratory infection incidence ~10-20% in deficient populations
  • K2 MK-7 has 72-hour plasma half-life vs 1-2 hours for MK-4; once-daily dosing is sufficient
  • Synergy hypothesis is largely preclinical; dedicated combination RCTs are limited
  • Daily dosing outperforms bolus dosing for immune and infection outcomes

Side-by-side

Attribute Rapamycin Vitamin D3 + K2
Category pharmaceutical supplement
Also known as Sirolimus, Rapamune cholecalciferol + menaquinone, D3/K2, vitamin D3 with MK-7
Half-life (hr) 62 360
Typical dose (mg) 6 0.05
Dosing frequency weekly (longevity protocols); daily for transplant indication daily with a fat-containing meal
Routes oral oral
Onset (hr) 1 24
Peak (hr) 2 168
Molecular weight 914.17 384.64
Molecular formula C51H79NO13 C27H44O (D3); C46H64O2 (MK-7)
Mechanism Binds FKBP12, and the resulting complex inhibits mTORC1, reducing protein synthesis and autophagy suppression downstream of nutrient and growth-factor signaling. D3 converts to calcidiol then calcitriol, activating the vitamin D receptor (VDR) to increase intestinal calcium absorption and modulate immune and bone gene transcription. K2 carboxylates osteocalcin and matrix Gla protein, directing calcium toward bone and inhibiting vascular calcification.
Legal status Prescription only (off-label for longevity) Dietary supplement (global)
WADA status allowed allowed
DEA / Rx Rx only (not a controlled substance) Not scheduled
Pregnancy Not recommended Recommended at standard doses for fetal bone development; consult clinician at higher doses
CAS 53123-88-9 67-97-0
PubChem CID 5284616 5280795
Wikidata Q410174 Q139347

Safety profile

Rapamycin

Common side effects

  • mouth ulcers (stomatitis)
  • acne-like rash
  • GI upset
  • altered lipid panel
  • delayed wound healing

Contraindications

  • active infection
  • severe hepatic impairment
  • planned surgery (delayed wound healing)
  • pregnancy
  • live vaccines within dosing window

Interactions

  • strong CYP3A4 inhibitors (ketoconazole, clarithromycin, grapefruit): substantially raises rapamycin levels, toxicity risk(major)
  • strong CYP3A4 inducers (rifampin, St John's wort): lowers rapamycin levels, reduced effect(major)
  • ACE inhibitors: increased risk of angioedema(moderate)
  • live vaccines: reduced vaccine efficacy due to immunosuppression(major)

Vitamin D3 + K2

Common side effects

  • GI upset at high doses
  • headache (rare)
  • hypercalcemia (only at sustained very high D3 doses)

Contraindications

  • hypercalcemia
  • sarcoidosis
  • active hyperparathyroidism
  • warfarin therapy (K2 component requires stable intake)

Interactions

  • warfarin: K2 component can affect anticoagulation; maintain stable intake and inform anticoagulation clinic(moderate)
  • thiazide diuretics: additive calcium retention; hypercalcemia risk with high-dose D3(moderate)
  • digoxin and calcium channel blockers: additive effects from D3-induced hypercalcemia(moderate)
  • glucocorticoids: reduced vitamin D efficacy and bone effects(moderate)
  • cholestyramine and orlistat: bind fat-soluble vitamins; separate dosing by 2 to 4 hours(moderate)

Which Should You Take?

Vitamin D3 + K2 comes out ahead for most readers on the criteria we weight: 3 catalogued goals, OTC dietary supplement, oral dosing, with a Tier-A outcome catalogued. Rapamycin is the right call when one of the conditionals below applies.

Edge case: If you want to avoid prescription-only, Vitamin D3 + K2 is the more accessible choice.

Default choice: Vitamin D3 + K2. Lower friction to source, a Tier-A evidence outcome catalogued, and broader goal coverage. Reach for Rapamycin only if your priority sits squarely in the goals it owns above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between Rapamycin and Vitamin D3 + K2?

Rapamycin and Vitamin D3 + K2 differ in category (pharmaceutical vs supplement), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, Rapamycin or Vitamin D3 + K2?

Rapamycin half-life is 62 hours; Vitamin D3 + K2 half-life is 360 hours.

Can you stack Rapamycin with Vitamin D3 + K2?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

Go deeper