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BiologicalX

Comparison

TUDCA vs Urolithin A

Side-by-side of TUDCA and Urolithin A. Every row below is pulled from the compound schema and will update as our data grows. For deeper reads, follow through to each compound page.

Effects at a glance

TUDCA

  • Bile-acid molecule (taurine-conjugated UDCA) with chemical chaperone activity at the endoplasmic reticulum
  • Established pharmaceutical use for cholestasis and primary biliary cholangitis at 500-750 mg/day
  • Reduces ER stress and stabilizes misfolded proteins; the mechanistic basis for emerging ALS / retinal applications
  • Modest improvements in NAFLD markers and insulin sensitivity at 500-1,750 mg/day in small trials
  • Mitochondrial protection signal in animal models drives the longevity-supplement positioning
  • Generally well-tolerated; mild GI effects are the main dose-dependent issue

Urolithin A

  • Gut-microbiome-derived metabolite of pomegranate and walnut ellagitannins
  • Roughly 40% of adults are 'urolithin producers' from dietary intake; ~60% are non-producers
  • Ryu 2016 (Nature Medicine) reported lifespan extension in C. elegans and muscle benefits in aged rodents
  • Andreux 2019 first-in-human trial (n=60) established safety and mitochondrial gene-expression upregulation
  • Singh 2022 (n=66, 4 months, 1000 mg/day) reported improved muscle endurance in older adults
  • Most human trial portfolio is Amazentis-funded; independent replication is thin

Side-by-side

Attribute TUDCA Urolithin A
Category supplement supplement
Also known as tauroursodeoxycholic acid, taurine-conjugated UDCA UA, Mitopure, ellagitannin metabolite
Half-life (hr) 4 17
Typical dose (mg) 500 500
Dosing frequency daily, divided into 2 doses with food daily, morning with food
Routes oral oral
Onset (hr) 1 2
Peak (hr) 2 4
Molecular weight 499.7 228.2
Molecular formula C26H45NO6S C13H8O4
Mechanism Bile-acid signaling via FXR/TGR5 receptors; chemical chaperone reducing ER stress and unfolded protein response; mitochondrial protection through reduced outer-membrane permeabilization. Induces mitophagy via potentiation of PINK1/Parkin signaling, leading to selective degradation of damaged mitochondria. Secondary anti-inflammatory effects via NF-kB modulation.
Legal status OTC dietary supplement (US); pharmaceutical in Italy and several Asian countries OTC dietary supplement (US GRAS 2018; EFSA Novel Food 2021)
WADA status allowed allowed
DEA / Rx OTC supplement OTC supplement (not scheduled)
Pregnancy Insufficient data for supplement use; UDCA used in cholestasis of pregnancy Insufficient data; not routinely recommended
CAS 14605-22-2 1143-70-0
PubChem CID 9848818 5488186
Wikidata Q418751 Q27101321

Safety profile

TUDCA

Common side effects

  • mild GI upset
  • diarrhea (dose-dependent)
  • constipation (rare)
  • nausea

Contraindications

  • complete biliary obstruction
  • pregnancy / lactation (insufficient supplement-dose data)
  • active GI disease without medical supervision

Interactions

  • cyclosporine, oral contraceptives, fat-soluble vitamins: modest absorption changes via altered bile-acid pool(minor)
  • phenylbutyrate: synergistic for ALS use (Relyvrio combination); consult clinician(moderate)

Urolithin A

Common side effects

  • mild GI upset (rare)
  • soft stools (rare)

Contraindications

  • pregnancy and lactation (insufficient data)
  • active chemotherapy (consult oncology)

Interactions

  • chemotherapy agents: theoretical interaction with mitochondrial-targeting agents; consult oncologist(moderate)

Which Should You Take?

TUDCA and Urolithin A score evenly on the criteria we weight (goal breadth, legal accessibility, evidence depth). The conditionals below should drive the decision more than any aggregate score.

  • If your priority is liver function, pick TUDCA.
  • If your priority is muscle hypertrophy, pick Urolithin A.
  • If your priority is healthspan extension, pick TUDCA.

Edge case: Half-lives differ materially (TUDCA ~4 hr vs Urolithin A ~17 hr). Urolithin A reaches steady state faster; TUDCA is easier to dial in if tolerability is uncertain.

Default choice: either is defensible. TUDCA edges out on goal breadth + legal accessibility; Urolithin A is the right call if your priority sits in the goals listed above.

This verdict is generated from each compound's schema (goals, legal status, evidence outcomes, dosing route). It updates automatically as our compound data evolves; the deeper read sits on each individual compound page.

Common questions

What is the difference between TUDCA and Urolithin A?

TUDCA and Urolithin A differ in category (supplement vs supplement), mechanism, and typical dosing. See the side-by-side table for full details.

Which has a longer half-life, TUDCA or Urolithin A?

TUDCA half-life is 4 hours; Urolithin A half-life is 17 hours.

Can you stack TUDCA with Urolithin A?

Stack compatibility depends on mechanism overlap, legal status, and individual response. Check each compound page for specific interactions and contraindications before combining.

Go deeper